Diefembaquia: planta venenosa

Re: OJO PLANTA MUY VENENOSA!

si nos ponemos en ese plan....no comas nada que te envenenas, pero toca todas las plantas que esas son buenas.

 

Re: OJO PLANTA MUY VENENOSA!

Toca todas las plantas ¿de la lista? Porque de hortigas a estoy "tocado"

 

Re: OJO PLANTA MUY VENENOSA!

pues que te aprovechen majo, si tienes la piel dura, mejor para ti eso que sales ganando. Pero hay mas gente en el foro.

 

Revisión muy util...

Querid@s!!!!

Ciertamente, estamos en presencia de una severa reacción alergica, un poco exagerada por supuesto para dar mas connotación al asunto, ademas los reportes médicos de revistas científicas de rigor no describen este florido y agresivo cuadro, tampoco sabemos las posibles patologías de base de esta persona que pudieron influir en tremenda reacción anafilactica... seamos realistas, nadie sabe a ciencia cierta lo que puede resultar un alergeno para cada individuo... solo por esto vamos a destruir a todas y cada una de las posibles sustancias que alguna vez inducieron un cuadro alergico grave a un paciente determinado en sus condiciones??? Explotemos el mundo pues!!

Es cierto que hay toxicidad, y para demostrarlo les dejo con referencias clave, en idioma ingles, por supuesto, toda la literatura científica de rigor esta en ese idioma, en el caso de Dieffenbachia se encuetra tambien Caladium y Philodendron y la cuestión es la presencia de cristales de oxalato de calcio, Y ENZIMAS PROTEOLITICAS que pudieran inducir cuadros de toxicidad moderada o leve , digase envenenamiento por ingesta o masticación de tallos u hojas de las mismas, si a esto le agregamos alergia, pues el caso tan grave en cuestion pudo ser posible pero de FORMA MUY AISLADA Y PARTICULAR, no generalicemos por favor!!! La vida es mas complejas y todos somos unicos...


En:
http://emedicine.medscape.com/article/1009003-overview
Se resume que pacientes con una historia de exposición oral a estas plantas antes mencionadas desarrollaron inflamación severa, salivación excesiva, disfagia (dificultad para comer ) y compromiso respiratorio, sintomas que no fueron usuales. en un estudio retrospectivo de 188 pacientes con expuestos, se determinó un a sintomatología mímina que resolvio con un tratamiento mínimo o sin tratamiento. Algunos pacientes con exposición dermica u ocular desarrollaron dermatitis o keratoconjuntivitis totalmente tratable Se describió una seria comlicación en un caso de una niña de 12.5 años, una fistula aortoesofageal resultante de la ingestion de las hojas de dieffenbachia en un case repost en el 2005, problema que fue resulto con cirugia sin mayores complicaciones!!

En:
http://emedicine.medscape.com/article/817016-overview
Se resume que en United States, la exposición a Philodendron and Dieffenbachia se encuentra entre las mas comunes repotadas por los poison control centers.
Segun el reporte anual 2007 de la American Association of Poison Control Centers' National Poison Data System (NPDS), se documentaron 7368 exposiciones a plantas con oxalatos, en la mayoría de los casos, lo oxalatos no solubles, producieron manifestations clinicas limitadas, ES EXTREMADAMENTE RARA UNA MORBILIDAD (ENFERMOS) O MORTALIDAD (MUERTOS) SIGNIFICATIVA. Se repotó un caso de muerto infantil por obstrucción de la vía aerea despues de la exposición a Dieffenbachia.
Este reporte plantea 1145 casos con sintomas leves, 81 moderados y solo 3 graves!! y ninguna muerte por exposición a los oxalatos...


Este resumen es mas concluyente:
http://informahealthcare.com/doi/abs/10.3109/15563659109025745

La ingestion de philodendron/dieffenbachia han sido asociados a desarrollo de irirtación intensa de la membrana mucosa (oral, conjuntiva), inflamción de la lengua , labios, paladar tongue, lips and palate. Aunque muchas publicaciones promueven su toxicidad, existen muy pocos casos que reporten una relación sustancial causa efecto entre la ingestion de las platas y la sintomatologia observada, Regional Poison Information Center (1991) realizo un estudio retrospectivo de 188 casos donde siempre se comprobo la ruptura de la integridad foliar, Ingirieron Philodendrons 67.5% and dieffenbachias el 32.5% of the cases. 72.8% de los casos en menores 4–12 meses. solo 2.1% (4) of the patients tuvieron sintomas (dieffenbachia-3; philodendron-1). en todos los casos los isntomas tuvieron lugar 5 min despues de la exposición y fueron de corta duracion y calsificados como leves, sin complicaciones o sintomas tardíos...

Si desean mas pueden leer aca, y buscar las revistas donde esta cada articulo, la fuente es totalmente confiable, Entrez PubMed es un servicio de busqueda bibliografica que reviza todoas las publicaciones científicas oficiales y de prestigio internacional, he puesto en Español algunos comentarios, buen provecho...

Hay toxicidad, la planta no es inocua, pero no hay que exgerar!!!! y no todos responderemos igual nunca a nada!!!!


http://www.ncbi.nlm.nih.gov/sites/entrez

Contact Dermatitis. 2005 Sep;53(3):172-3.
Contact dermatitis due to Dieffenbachia.
Sanchez-Morillas L. Servicio de Alergia, Clinica Puerta de Hierro, Madrid, Spain. lsanchezmorillas@hotmail.com

J Pediatr Surg. 2005 Jun;40(6):e29-31.
Aortoesophageal fistula--an unusual complication of esophagitis caused by Dieffenbachia ingestion.
Snajdauf J, Mixa V, Rygl M, Vyhnánek M, Morávek J, Kabelka Z.
Aortoesophageal fistula (AEF) is a rare and mostly lethal cause of upper gastrointestinal bleeding in children. We report a successful outcome of surgical treatment after resuscitation of a girl aged 12 1/2 years with AEF as a complication of esophagitis caused by Dieffenbachia ingestion. The girl ate a leaf of Dieffenbachia picta in a suicidal attempt. After 5 weeks of medical treatment of esophagitis, small blood-stained vomitus and melena appeared. Within a few hours, it was followed by a massive gastrointestinal bleeding leading to exsanguination. The girl survived despite the pitfalls in clinical presentation and errors in diagnostic approach and treatment. Emergency exploration of cervical esophagus, followed by laparotomy and thoracotomy, was performed in attempt to find the source of bleeding and to control it. Aortoesophageal fistula was located between the ascending aorta near the origin of the brachiocephalic trunk and thoracic esophagus. Resection of the fistula led to prompt circulatory stabilization. The common houseplant, Dieffenbachia picta, causes edematous swelling of mucus membranes when chewed. Dieffenbachia-caused esophagitis is very rare and this etiology of AEF has not been reported in children. (NIÑA SOBREVIVE)

J Chin Med Assoc. 2004 Feb;67(2):93-8.
Ocular injuries from plant sap of genera Euphorbia and Dieffenbachia.
Hsueh KF, Lin PY, Lee SM, Hsieh CF.
Department of Ophthalmology, Taipei Veterans General Hospital, Taipei, Taiwan, ROC.
Clinical findings in 3 cases of ocular injuries caused by the milky latex from Euphorbia tirucalli and Dieffenbachia sequine were reported. The initial symptoms of all patients were burning pain with subsequently blurred vision. Visual acuity was reduced between 6/30 and 6/20. Euphorbia sap caused punctate erosion, microbullae and Descemet's folds; while Dieffenbachia sap induced conjunctival chemosis and fine blue crystals in the stroma. Ocular symptoms developed in 5 to 18 hours despite immediately copious irrigation. Supportive treatment resulted in a full recovery without sequalae over 1 week. Wearing eye protection and washing the exposed areas with soap and water are advised while handling such plants. ( RECUPERACION SIN SECUELAS!!!!)

Toxicon. 2004 May;43(6):729-35.
Ability of eugenol to reduce tongue edema induced by Dieffenbachia picta Schott in mice.
Dip EC, Pereira NA, Fernandes PD.
Laboratório de Farmacologia da Inflamação e do Oxido Nítrico, Departamento de Farmacologia Básica e Clínica, ICB, Universidade Federal do Rio de Janeiro, Caixa Postal 68016, 21944-970 Rio de Janeiro, Brazil.
Dieffenbachia picta Schott (Araceae), known in Brazil as "comigo-ninguém-pode" is an ornamental plant with toxic properties. Its juice, when chewed, causes a painful edema of the oral mucous membranes, buccal ulcerations and tongue hypertrophy. This acute inflammation sometimes becomes severe enough to produce glottis obstruction, respiratory compromise and death. Eugenol (4-alil-2-metoxiphenol), the essential oil extracted from Caryophyllus aromaticus (Myrtaceae) is widely used in odontology. In this study, our objective was to standardize, in mice, a measurable methodology for the tongue edema induced by the topical application of the D. picta stem juice; evaluate the effects of eugenol in this model and compare the results with emergency treatment used in hospitals. Our results show that in spite of a small increase in edema a few minutes after administration, emergency treatment reduced by 70% the overall edema. When compared with the combination of the above drugs, eugenol, even at the smallest dose of 5 microg/kg, regardless of the chosen administration route, or the moment the treatment began, presents better results in the reduction and inhibition of the tongue edema induced by the D. picta juice. ( EL EUGENOL PARA TRATAR LOS CASO AGUDOS)

J Emerg Med. 2003 Nov;25(4):391-7.
* J Emerg Med. 2004 May;26(4):491.
Acute airway compromise after brief exposure to a Dieffenbachia plant.
Cumpston KL, Vogel SN, Leikin JB, Erickson TB.
Dieffenbachia is a common domestic plant. Oral contact with the plant usually is associated with minimal consequences. However, chewing on the stem or the leaf of the Dieffenbachia can result in painful oropharyngeal edema and the inability to speak or handle secretions. Airway compromise has been reported only in a pediatric ingestion. We report a case of an adult who bit into the stem of the plant thinking it was sugar cane. He instantly spit out the remaining stem and despite this brief exposure, oropharyngeal edema developed refractory to medicinal therapy, requiring surgical airway management. The exact mechanism of edema is not known; therefore, methods of treatment are variable. We recommend caution when presented with a patient exposed to Dieffenbachia. Even the patient who initially seems stable may have an airway that will quickly deteriorate. ( ESTE CASO CHUPO EL TALLO COMO CAÑA DE AZUCAR Y DEFINITIVAMENTE NO LE FUE TAN BIEN)

Vet Hum Toxicol. 2003 Oct;45(5):233-9.
Accidental fatal poisoning of a dog by Dieffenbachia picta (dumb cane).
Loretti AP, da Silva Ilha MR, Ribeiro RE.
Sector of Veterinary Pathology, Department of Veterinary Clinical Pathology, Faculty of Veterinary Medicine, Federal University of Rio Grande do Sul, Av. Bento Gonçalves, 9090, Bairro Agronomia, PO Box 15094, Porto Alegre, RS, Brazil, CEP 91540-000.
A single case of accidental fatal poisoning by Dieffenbachia picta, (dumb cane) (Araceae) in a 9-y-old female Poodle is described. Clinical signs included severe, locally extensive erosive/ulcerative glossitis accompanied by marked dyspnea of acute onset. The animal did not respond to emergency procedures to relieve severe respiratory distress; fatal asphyxiation ensued from edema of the glottis a few hours after the first clinical signs were observed. According to the owner, the dog had access to a tall, potted dumb cane plant and chewed the thick stem of the plant intensely. The owner did not authorize a necropsy of the animal. The diagnosis of Dieffenbachia picta poisoning was based on the history of accidental consumption of dumb cane and clinical signs. A comparison of this single case with other reports of dumb cane poisoning suggests that dogs poisoned by Dieffenbachia species usually recover uneventfully with conservative management and that death from asphyxiation is a rare but possible consequence of this intoxication. Severe edematous swelling of the glottis with occlusion of the larynx airway passage can occur in those cases in which large amounts of sap are quickly squeezed from the plant during intense chewing. Death would occur if owners do not seek veterinary care immediately and if emergency procedures are not instituted on time. ( ESTE PERRITO POODLE SI SE MURIO)

Soc Hist Med. 2002 Dec;15(3):481-504.
A darker shade of green: medical botany, homeopathy, and cultural politics in interwar Germany.
Kenny MG.
Department of Sociology and Anthropology, Simon Fraser University, Burnaby, Canada. Michael_kenny@sfu.ca
In 1941 a proposal was made to Nazi SS Reichsführer, Heinrich Himmler, that extracts of a South American plant, Dieffenbachia seguine, might be used for the mass sterilization of racially undesirable war prisoners. The proposal was based on published animal fertility research conducted by Dr Gerhard Madaus, co-founder of a firm that produced and marketed natural medicinals. His fertility experiments were part of a broader series aimed at evaluating the scientific validity of ethnobotanical folk-knowledge. This article traces the historical background to the Madaus research: first, the role of homeopathy in the introduction of Dieffenbachias to western medicine; secondly, the social context of German 'alternative' medicine in the interwar period; and finally, the role of Madaus himself, whose homeopathically-oriented research on botanical medicinals inadvertently initiated the chain of events described here. ( ESTE ARTICULO REFIERE UNA VEZ MAS LO HORRIBLE DEL FASCISMO QUE UTILIZO LA PLANTA COMO POSIBLE AGENTE PARA INDUCIR INFERTILIDAD )

: Vet Hum Toxicol. 2002 Oct;44(5):298-300.
Related Articles, Links

Contrary to the literature, vomiting is not a common manifestation associated with plant exposures.

Krenzelok EP, Mrvos R, Jacobsen TD.

Pittsburgh Poison Center, Children's Hospital of Pittsburgh, School of Pharmacy, University of Pittsburgh, PA, USA.

A review of clinical toxicology references and the contemporay literature illustrates that vomiting is reported as a nearly universal symptom associatedwith exposure to cultivated and wild plants. However, the literature fails to put thesymptom of vomiting into a clinical perspectivewith regard to its frequency of occurrence. To clarify this issue a retrospective review of plant exposures was conducted. AAPCC TESS 1997-1999 was queried electronicaly to identify and extract all plant ingestion exposures where vomiting was documented as being related to the exposure. All exposures with documented ipecac syrup use were excluded. Data were stratified through the use through the use of a relational database. Plant ingestion exposures accounted for 229,538 reports and vomiting was reported in 5,917 (2.6%) of exposures. Vomiting was attributed as a related symptom in 753 different plants. It was reported once in 323 plants, 1-3 times in 495 plants (65.7%), and < or = 10 times in 657 (87.4%) of all plants in the database. Ten plants accounted for 32.3% of the reports of vomiting Philodendron, Spathiphyllum, Narcissus, Dieffenbachia, Phytolacca, Epipremnum, Euphorbia, Eucalyptus, Ficus, Hedera) and represented 1.3% of the plants associated with vomiting. Even with the most common plant exposures, vomiting is not a frequent adverse event. (CONTRARIO A LO QUE SE PIENSA EL VOMITO NO ES UN SINTOMA DE INTOXICACIÓN POR ESTAS PLANTAS)

Hum Exp Toxicol. 2001 Apr;20(4):189-92.
Tagwireyi D, Ball DE.
Department of Pharmacy, Medical School, University of Zimbabwe, Avondale, Harare.
The Araceae family of plants is the major cause of symptomatic plant ingestions in some developed countries (Dieffenbachia and Philodendron) and in Zimbabwe (Elephant's Ear), especially in children. A retrospective case series was carried out to evaluate the management of poisoning due to Elephant's Ear at the largest referral hospital in Zimbabwe for the period January 1995-December 1999. The study revealed inappropriate use of antibiotics, atropine, and antihistamines in the treatment of Elephant's Ear poisoning. This article also reviews the management of poisoning due to the Araceae family of plants as exemplified by Elephant's Ear. There is a need to educate health care workers on the clinical management of Elephant's Ear poisoning especially in developing countries where there are limited resources.
( MAS REFERENCIAS DE TOXICIDAD)


Vet Hum Toxicol. 1999 Oct;41(5):335-8.
Dieffenbachia species exposures: an evidence-based assessment of symptom presentation.
Pedaci L, Krenzelok EP, Jacobsen TD, Aronis J.
School of Pharmacy, University of Pittsburgh, PA, USA.
The literature often ascribes significant morbidity to Dieffenbachia exposures. However, poison center experience suggests a discordant view from that presented in the literature. This project determined if the symptoms described in the literature were consistent with clinical practice. To profile the symptoms reported in the literature, an extensive literature search using Medline and Webspirs (http:/(/)infoshare.library.pitt.edu/webspirs /webspirs.htm) was conducted to identify publications that described Dieffenbachia toxicity. Standard toxicology references and medical botany texts found in poison centers were used to profile symptoms in the tertiary literature; and an Internet search using the Savvy Search program (http:/(/)www.guaraldi.cs.colostate.edu:20 00) to search simultaneously all other search engines. The symptom information was compared to AAPCC TESS (1993-1996) symptom data reported on Dieffenbachia exposure reports. Twenty-three reference sources profiled the literature perspective on Dieffenbachia exposures. AAPCC TESS contained 10,796 Dieffenbachia exposures, and 34.7% of the reports documented symptoms. Oral irritation was reported in 18.2% of the reports and 92.6% of the queried literature listed this symptom (18.2%/92.6%). Additionally dermal pain = 8.7%/63.0%; vomiting = 2.6%/18.5%; erythema = 2.5%/5.3%; throat irritation = 2.3%/22.2%; dermal edema = 2.2%/3.7%; pruritus = 2.1%/7.4%; ocular irritation = 1.7%/44.4%; rash = 1.2%/29.6%; cough/choke = 1.1%/3.7%; loss of speech = 0%/29.6%; salivation = 0%/29.6%; respiratory obstruction = 0%/48.1%; death = 0%/11.18%. When multiple symptoms occurred the most common toxidrome was oral and throat irritation that occurred in 2.9% of the AAPCC TESS reports. There is similarity between the literature and the clinical experience with regard to the irritant properties of Dieffenbachia species, but there is also significant disparity because the literature fails to put the symptoms into a clinical frequency perspective. In general, the literature portrays Dieffenbachia exposures associated with more morbidity and mortality than what was reported in the AAPCC TESS clinical practice data. ( ARTICULO IMPORTANTISIMO DONDE SE REFIERE UNA DISCORDANCIA ENTRE LA LITERATURA QUE ASEGURA UNA GRAN TOXICIDAD NO DEMOSTRANDOSE ESTO EN LA PRACTICA MEDICA DIARIA)

Indian J Biochem Biophys. 1998 Dec;35(6):358-63.
Related Articles, Links

A cysteine protease of Dieffenbachia maculata.

Chitre A, Padmanabhan S, Shastri NV.

University Department of Biochemistry, Nagpur University, India.

Plants of the genus Dieffenbachia, very popular as indoor ornamental plants, are known for their toxic as well as therapeutic properties. Their toxic manifestations have been partly attributed to their proteolytic activity. The work described in the present paper shows that stem leaves and petiole of Dieffenbachia maculata Schott, a commonly grown species, contain significant proteolytic activity, different parts showing different types of protease activities. Stem showed the highest enzyme activity and this protease was purified about 55 fold by solvent precipitation, gel filtration and ion exchange chromatography. The enzyme has a relative molecular mass of 61 kDa as determined by SDS-PAGE and has an optimum pH of 8.0 and optimum temperature of 50 degrees C. Effects of various substrates, inhibitors and activators indicate that the enzyme is a cysteine protease with leucylpeptidase activity. ( SE EXPLICAN LOS MECANISMOS DE TOXICIDAD ASOCIADOS A UNA PROTEASA, ENZIMA QUE DEGRADA LAS PROTEINAS, IMPORTANTE PARA LA LIBERACION DE LOS OXALATOS)



Tierarztl Prax Ausg K Klientiere Heimtiere. 1998 Nov;26(6):404-7.

[Extensive stomach ulcers due to Dieffenbachia intoxication in a cat]
[Article in German]
Müller N, Glaus T, Gardelle O.
Veterinär-Medizinischen Klinik, Universität Zürich.
Extensive gastric ulceration was diagnosed endoscopically and histologically in a two-year-old cat with haematemesis, anorexia and severe dehydration. Based on history (previous ingestion of leaf pieces of Dieffenbachia) and exclusion of another cause, intoxication with Dieffenbachia was diagnosed. The toxical effect of Dieffenbachia on mucous membranes is probably explained by ejection of calcium oxalate crystals leading to injury of mast cells and a massive histamine release. Supportive treatment including electrolyte infusions, ranitidine, sucralfate, and forced feeding using an endoscopically placed gastrostomy tube resulted in quick clinical recovery of this cat. ( ESTE GATO DESARROLLO ULCERAS PERO SE SALVO)

J Eur Acad Dermatol Venereol. 1998 Jan;10(1):87-9.
Irritant contact dermatitis due to Dieffenbachia s.p.p.
Corazza M, Romani I, Poli F, Virgili A.
( OTRA REFERENCIA DE DERMATITIS POR CONTACTO)


Vet Hum Toxicol. 1997 Aug;39(4):248-9.
Plant exposures ... a national profile of the most common plant genera.
Krenzelok EP, Jacobsen TD.
Pittsburgh Poison Center, Children's Hospital of Pittsburgh, School of Pharmacy, University of Pittsburgh, PA 15213, USA.

Plant exposures are the 4th most common cause of poisoning and 85% of those exposures involve the pediatric population. The large number of plant-related exposures and the lack of knowledge about plant toxicity has led to plant paranoia and considerable educational efforts to reduce the number of exposures. These efforts are often dictated by misconceptions and folklore. American Association of Poison Control Centers (AAPCC) data on all plant exposures for the years 1985-1994 were electronically analyzed by plant genus to establish a frequency distribution of all plant exposures in the United States. A total of 912,534 plant exposure cases were analyzed to tabulate the top 100 plant exposures. Philodendron spp were the most common exposures, followed by Dieffenbachia spp, Euphorbia spp, Capsicum spp and Ilex spp. Plant exposures are very common and poison information centers devote significant clinical service and educational effort to manage these exposures and enhance the public's awareness. Awareness of specific plant species and exposure frequency in a poison center region can serve as a basis for staff education and as the cornerstone for the development of appropriate information in poison prevention and education brochures. This can direct better utilization of poison center financial resources.( UN ANALISIS DEMUESTRA QUE Philodendron ES MAS COMUN EN INTOXICACIONES)

Pediatr Rev. 1997 May;18(5):162-8.
Poisonous plants: when they are a threat to children.
Lawrence RA.
University of Rochester School of Medicine, Rochester, NY, USA.
Plant toxic to humans abound in home and yard as well as open fields and woods. Only experienced botanists and serious students of edible vegetation can distinguish safely what is edible, inedible, or simply toxic. Parents should be instructed to protect their children from truly toxic household plants and train their children never to eat a plant not provided to them by someone trustworthy as edible. If a child or an adult consumes a possibly toxic plant, the regional poison center should be contacted for identification and management instructions. As with all potential poisonings, the sooner the management is initiated, the better the outcome. Adults have been seriously poisoned by confusing a toxic plant with something that they thought was edible (eg, pokeweed root for horseradish). The pediatrician's office should be a model, with only nontoxic plants on display-no dieffenbachia, peace lily, pothos, or philodendron, for example. ( EDADES PEDIATRICOS OJO CON ESTO)

Br J Ophthalmol. 1997 Feb;81(2):168-9.
Diagnosis of Dieffenbachia induced corneal injury by confocal microscopy.
Chiou AG, Cadez R, Böhnke M.
( DAÑO CORNEAL POR LA PLANTA)



Vet Hum Toxicol. 1996 Aug;38(4):289-98.
Plant exposures: a state profile of the most common species.
Krenzelok EP, Jacobsen TD, Aronis JM.
Pittsburgh Poison Center, Children's Hospital of Pittsburgh, PA 15213, USA.
Plant exposures are the fourth most common cause of poisoning and 86% of those exposures involve the pediatric population. The large number of plant-related exposures and the lack of knowledge about plant toxicity has led to plant paranoia and considerable educational efforts to reduce the number of exposures. These efforts are often dictated by misconceptions and folklore. AAPCC TESS data on all plant exposures for the years 1985-1994 were electronically analyzed by plant species and state to establish a frequency distribution in the US. Aggregate national data was also tabulated. A total of 912,534 plant exposures were analyzed to tabulate the top 30 plant exposures for each state. Philodendron species were the most common exposures, followed by Dieffenbachia species, Euphorbia pulcherrima, Capsicum annuum and Ilex species. There were considerable differences between states relating to indoor vs outdoor plants and native vs introduced varieties. Plant exposures are common and poison information centers devote significant clinical service and educational effort to manage them and to enhance public awareness. Recognition of specific plant species and exposure frequency in a poison center region can be a basis for staff education and the development of appropriate poison prevention education brochures. This can direct better utilization of poison center's resources.
( SIGUEN LOS POBRES FILO EN PRIMER LUGAR!!)

Schweiz Med Wochenschr. 1996 Jun 22;126(25):1085-98.
[Serious plant poisonings in Switzerland 1966-1994. Case analysis from the Swiss Toxicology Information Center]
[Article in German]
Jaspersen-Schib R, Theus L, Guirguis-Oeschger M, Gossweiler B, Meier-Abt PJ.
Schweizerisches Toxikologisches Informationszentrum (STIZ), Zürich.
AIM: To analyze the types, frequency and severity of plant poisonings in Switzerland over 29 years. METHODS: Retrospective analysis of severe poisonings with toxic plants reported to the Swiss Toxicological Information Center (STIC). Assessment of the causality, severity of symptoms and the types of plants involved. RESULTS: During the period under review the STIC registered 24 950 cases of contact with or ingestion of toxic plant material. In 99.4% of all cases the clinical course was either unknown, asymptomatic or associated with only minor symptoms (no hospitalization). Severe plant poisonings occurred in 152 cases. Detailed analysis was possible in 135 cases (23 children, 112 adults) including 5 lethal cases (all adults). The 24 plants involved produced the following severe symptoms: Atropa belladonna (42 cases): anticholinergic syndrome (42), acute psychosis (33), convulsions (2), coma (2). Heracleum mantegazzianum (1: severe photodermatitis (1. Datura stramonium (17): anticholinergic syndrome (17), psychosis (12), coma (2). Dieffenbachia (11): severe stomatitis (, corneal lesions (3). Colchicum autumnala (10): diarrhea (10), liver necrosis (9), fatal multiorgan failure (2). Veratrum album (: bradycardia ( < or = 40/min) (6), shock (5). Aconitum napellus (4): tachyarrhythmia (2), AV-block II/III (2). Aesculus hippocastanum (3): allergy (3), anaphylactic shock (2). Hyoscyamus niger (3): anticholinergic syndrome (3). Ricinus communis (3): diarrhea (3), toxic megacolon (1). Oenanthe crocata (2): convulsions (1), lethal coma (1). Taxus baccata (2): tachyarrhythmia (1), fatal asystole (1). Further single cases with severe poisonings were observed with Arum maculatum, Asarum europaeum, Chrysanthemum vulgare, Cyclamen persicum, Datura suaveolens, Glycyrrhiza glabra, Laburnum anagyroides, Lycopodium, Narcissus pseudonarcissus (lethal aspiration), Nerium oleander, Senecio vulgaris and Vicia faba. CONCLUSIONS: Potential and real intoxications with plant materials occurred in 7.2% of all cases registered at the STIC. However, among all plant cases only 0.6% were severe intoxications requiring hospitalization. Although severe plant intoxications are rare events, a small number of specific plants appear to be mainly responsible for continued serious plant poisoning in Switzerland. The present study has identified the plants with the highest toxicological risks and provides a data base for more rational prevention, diagnosis and treatment of plant poisoning cases in the future. ( PARA NUESTRA Dieffenbachia SOLO ULCERAS CORNEALAES Y ESTOMATITIS)


Klin Oczna. 1996;98(4):311-4.
[Harmful impact of Diffenbachia sap on the anterior segment of the eye]
[Article in Polish]
Matysik A.
II Kliniki Okulistyki AM w Lublinie.
The author presents a case of a woman with conjunctival and corneal changes caused by the drop of the sap form a decorative home plant Dieffenbachia. The patient complained of pain, burning and slight worsening of vision Conjunctival swelling, lesions of corneal epithelium with a number of minute foreign bodies of a needle-like shape in cornea were observed. The mechanism of the Dieffenbachia toxicity as well as the management of the injuries are discussed.
( UN REPORTE DE CASO DE LESION OCULAR)

Br J Ophthalmol. 1995 Jan;79(1):98-9.
Crystalline keratopathy from Dieffenbachia plant sap.
Seet B, Chan WK, Ang CL.
Singapore National Eye Centre.
( OTRO REPORTE DE CASO DE LESION OCULAR)


Adv Space Res. 1994 Nov;14(11):99-103.
Toluene removal from air by Dieffenbachia in a closed environment.
Porter JR.
Department of Biological Sciences, Philadelphia College of Pharmacy and Science, PA 19104, USA.
Higher plants are likely to play a major role in bioregeneration systems for food, air and water supplies. Plants may also contribute by the removal of toxic organic substances from the air of a closed environment. Dieffenbachia amoena plants were exposed to 0 to 1.2 x 10(6) micrograms toluene m-3 at light intensities of 35 and 90 micromoles m-2 s-1 in sealed chambers. Toluene removal, photosynthesis and respiration were measured. An increased light intensity increased the rate of toluene removal five-fold over the rate at the lower intensity; the kinetics suggest active regulation by the plant. The removal rate saturated at 2700 micrograms toluene h-1 at the lower intensity and failed to saturate at the higher intensity. Toluene exposure inhibited photosynthesis and respiration only transiently and without correlation to toluene concentration. These plants can act as efficient scavengers of toluene in a contaminated environment. ( SIN EMBARGO MIEREN QUE MARAVILLA, ESTA PLANTA PUEDE REMOVER GRANDES CANTIDADES DE TOLUENO DE NUESTRA ATMOSFERA, LO QUE REDUCE LA CONTAMINACIÓN AMBIENTAL, UN MARAVILLOSO BIOREGENERADOR)

Oral Surg Oral Med Oral Pathol. 1994 Nov;78(5):631-3.
Injury to the oral mucous membranes caused by the common houseplant, dieffenbachia. A review.
Gardner DG.
Division of Oral Pathology and Oncology, University of Colorado School of Dentistry, Denver.
The common houseplant, dieffenbachia, causes painful edematous swelling of the oral mucous membranes when chewed. This property, which is well known to the staffs of poison control centers, can be dangerous to the unwary or to victims of practical jokes. The microscopic features of the injury are those of acute inflammation, but the nature of the plant's toxicity remains controversial. The numerous needles (raphides) of calcium oxalate, which are contained in specialized cells (idioblasts) in the plant, and proteases have both been implicated. When stimulated, the idioblasts fire the raphides with some force for a distance of two to three cell lengths. One possibility is that the local toxicity of dieffenbachia may be caused by a combination of this mechanical release of the raphides, which results in traumatic injury, and the effects of a chemical toxin carried on their surfaces and in their grooves.
( DAÑO DE LA CAVIDAD ORAL, INFLAMACIÓN, SALIVACIÓN)

Wiad Lek. 1994 Mar;47(5-6):220-1.
[A case of Dieffenbachia poisoning]
[Article in Polish]
Maj J.
Oddziału Laryngologicznego Wojewódzkiego Szpitala Specjalistycznego w Tychach.
A case is presented of poisoning with, even more frequently cultivated in flats, Dieffenbachia. The mechanism of toxic effect of the juice of this plant is discussed. Prophylactic and therapeutic guidelines are presented.
( OTRO CASO CON LESIONES ORALES)

J Fla Med Assoc. 1992 Nov;79(11):760-1.
The dieffenbachia plant. Case history.
Pamies RJ, Powell R, Herold AH, Martinez J 3rd.
Division of General Internal Medicine, Mt. Sinai Medical Center, Cleveland, Ohio.
When ingested, the common house plant dieffenbachia can lead to significant toxicity and possibly death if timely medical attention is not forthcoming following initial exposure. The deleterious effects should not be overlooked especially when children are involved who, naturally curious, may bite into this innocuous looking plant. Although the patient in this case was a type II diabetic on insulin, neither hypo- nor hyperglycemia developed. There was no other significant electrolyte imbalance despite the acute stressor placed on him.

( UN CASO GRAVE, DIABETICO, LO QUE LES DECIA DE LAS ENFERMEDADES DE BASE)

J Am Vet Med Assoc. 1992 May 1;200(9):1329-31.
Evaluation of selected plants for their toxic effects in canaries.
Arai M, Stauber E, Shropshire CM.
Department of Veterinary Clinical Medicine and Surgery, College of Veterinary Medicine, Washington State University, Pullman 99164-6610.
Leaves or fruit from 14 plants considered to be toxic to pet birds were administered by gavage to 15 pairs of canaries (Serinus canaria). Each bird was given 0.12 to 0.70 g of plant material. One pair served as a control and was given distilled water. The plant materials were flash-frozen in liquid nitrogen, pulverized, and resuspended in deionized water for administration. Of the plants tested, 5 (oleander, lupine, foxglove, yew leaves, and dieffenbachia) were considered highly toxic and were associated with acute death of birds. The remaining plant samples caused no, or only transient, clinical illness.
( LOS CANARIOS SI SE MURIERON... LAS AVES!! )

J Toxicol Clin Toxicol. 1991;29(4):485-91.
Philodendron/dieffenbachia ingestions: are they a problem?
Mrvos R, Dean BS, Krenzelok EP.
Pittsburgh Poison Center, Children's Hospital of Pittsburgh, PA 15213-2583.
Ingestions involving the philodendron/dieffenbachia members of the Arum family are allegedly associated with the development of intense irritation of mucous membranes, resulting in swelling of the tongue, lips and palate. Although numerous literature citations promote their toxicity, there are few case reports which substantiate a cause-effect relationship between ingestion and resultant symptomatology. To assess the toxic manifestations associated with the ingestion of these plants, a retrospective review of such cases reported to a Regional Poison Information Center was performed. 188 cases were identified and the integrity of the leaf had been broken in all cases. Philodendrons accounted for 67.5% and dieffenbachias for 32.5% of the cases. 72.8% involved children aged 4-12 months. Only 2.1% (4) of the patients were symptomatic (dieffenbachia-3; philodendron-1). In all cases, the symptoms occurred within 5 minutes of the exposure and were of short duration and the outcome was classified as minor. In this 24 month senses of 188 exposures, severe oral complications and the delayed development of symptoms were not observed.
( ESTE RESUMEN LO DISCUTÍ AL INICIO)

Cesk Pediatr. 1989 May;44(5):305-7.
[Dieffenbachia and toxicological problems]
[Article in Czech]
Jícha J, Vostatek M, Toman J.

Cesk Oftalmol. 1988 Nov;44(6):453-5.
[Dieffenbachia and the eye]
[Article in Czech]
Vácha J, Bodnár M

Ugeskr Laeger. 1984 Dec 10;146(50):3927-8.
[Eye injuries may be due to the potted plant Dieffenbachia]
[Article in Danish]
Ottosen CO, Irgens-Møller L.

Br Dent J. 1987 Jun 20;162(12):467-8.
Oral ulceration after contact with the houseplant Dieffenbachia.
Evans CR.

Emerg Med Clin North Am. 1984 Aug;2(3):553-62.
Common toxic plant ingestions.
Geehr E.
tHis article focuses on some of the more common dangerous plant and mushroom ingestions, discussing toxic components, early symptoms, and treatment. Among the plants considered are oleander, foxglove, hemlock, dieffenbachia, and Amanita phalloides. Details concerning recognition of toxic species are intentionally omitted in an effort to avoid potentially dangerous errors in identification.

Cesk Otolaryngol. 1982 Aug;31(4):239-41.
[Damage to the deglutition tract by Dieffenbachia]
[Article in Czech]
Navrátilová A, Mikolaj M.

J Ethnopharmacol. 1982 May;5(3):293-302.
Dieffenbachia: uses, abuses and toxic constituents: a review.
Arditti J, Rodriguez E.

Singapore Med J. 1977 Sep;18(3):176-7.
External eye allergy from sap of Dieffenbachia picta.
Lim KH

Klin Monatsbl Augenheilkd. 1977 Jan;170(1):128-30.
Related Articles, Links

[Ocular involvement due to the juice of Dieffenbachia plants (author's transl)]
Egerer I.
Upon breaking the stem or the branches of a Dieffenbachia plant juice is released in an explosive manner and may thus involve the eye. A myriad of fine calcium-oxalate crystals penetrate into all layers of the cornea, and an iritis can develop. The latter ceases within 6 days, the corneal cristals vanish within about three weeks.

Dtsch Gesundheitsw. 1971 Jan 26;26(2):73-6.
[Eye injury by the juice of the plant "Dieffenbachia seguine"]
[Article in German]
Riede B.

( MUCOSA ORAL Y OCULAR, LA MAS REPORTADA CON DAÑOS)

Derm Beruf Umwelt. 1986 Jul-Aug;34(4):93-101.
Related Articles, Links

[Skin and mucous membrane reactions caused by Dieffenbachia species house plants]
[Article in German]
Ippen H, Wereta-Kubek M, Rose U.
A survey, based on 60 essays in the relevant medical and botanical literature, reviews the erosive effects, on skin and mucous membranes, caused by contact with plants of the Dieffenbachia species. Although these common house plants are the most frequent cause of skin and mucous membrane damage by flora and fauna, the pathogenesis of these rapidly occurring caustic actions has not been satisfactorily explained. The assumption that "injection" of proteolytic enzymes into healthy tissue by "shooting cells" (raphid-idioblasts), full of calcium oxalate needles, is responsible, must be viewed as the most readily acceptable explanation. Unfortunately, this hypothesis can not be regarded as proven.

( SE INTENTA EXPLICAR LA ACCIÓN CAUSTICA DE LA PLANTA, ASUMIENDO QUE LA SAVIA DE LA MISMA CONTIENE CELULAS LLENAS DE CRISTALES DE OXALATO DE CALCIO EN FORMA DE FINAS AGUJAS QUE INYECTAN ESAS ENZIMAS PROTEOLITICAS DE LAS QUE LES HABLE EN EL TEJIDO SANO)

J Toxicol Clin Toxicol. 1985;23(2-3):79-90.
Observations on the idioblasts of Dieffenbachia.
Rauber A.
The toxicity of the Arum genus, especially the Dieffenbachia, has long been a topic of controversy. Some believe that the injury inflicted is primarily mechanical, the result of needle like crystals of calcium oxalate (raphides) found in the stems and leaves. Others cite evidence supporting the existence of enzymatic and allergic injury. This paper describes a hitherto neglected third factor, specialized cells which can forcibly shoot the raphides and which may be necessary for the toxic effect since raphide containing plants devoid of these cells are not toxic. The effects of physical and chemical factors on the behavior of these cells are described

( ACA SE DESCRIBE MEJOR EL MECANISMO DE TOXICIDAD, DONDE SE HABLA DE DAÑO MECANICO POR LOS CRISTALES, DAÑO POR ENZIMAS Y ALERGIAS!!!!!!!!!)

Toxicol Appl Pharmacol. 1981 May;58(3):444-51.
Study of Dieffenbachia-induced edma in mouse and rat hindpaw: respective role of oxalate needles an trypsin-like protease.
Kuballa B, Lugnier AA, Anton R.

( LAS AGUJAS DE OXALATO Y LA ENZIMA QUE DEGRADA PROTEINAS)



J Ethnopharmacol. 1984 Dec;12(3):293-303.
Investigations on Dieffenbachia amoena Gentil. I: Endocrine effects and contraceptive activity.
Costa de Pasquale R, Ragusa S, Circosta C, Forestieri AM.
Contraceptive activity and the correlated endocrine alterations of an aqueous extract of the leaves of Dieffenbachia amoena were investigated. In female Wistar rats the extract, given for 4 days during the estrous cycle, induced significant variations of luteinizing hormone (LH), follicle-stimulating hormone (FSH), prolactin (PRL) progesterone and 17 beta-estradiol levels. It also determined a reversible interruption of the estrous cycle with consequent temporary inhibition of ovulation.
( OJO SE DEMUESTRA EN RATONES EFECTOS CONTRACEPTIVOS POR EFECTOS ENDOCRINOS EN LA SECRECION DE HORMONAS)


Res Front Fertil Regul. 1982 Jun;2(1):1-16.
Current status of plant products reported to inhibit sperm.
Farnsworth NR, Waller DP.
PIP: This report reviews research on plant-derived agents that prevent sperm production if taken orally by the male or that incapacitate or kill sperm on contact if used vaginally by the female. It would be of great value to develop fertility inhibitors that are totally selective for reproductive systems and enzymes, and there is a possibility that a plant-derived drug may have this effect. Plants that have been studied for their fertility inhibiting effects in the male include: Aristolochia indica L. (Aristolochiaceae); Azadirachta indica A. Juss (Meliaceae); Balanites roxburghii Planch. (Zygophyllaceae); Calotropis procera (Ait) R.Br. (Asclepiadaceae); Carica papaya L. (Caricaceae); Catharanthus roseus (L.) G. Don (Apocynaceae); Dieffenbachia seguine (Jacquin) Schott. (Araceae); Ecaballium elaterium A. Richard (Cucurbitaceae); Gossypium species (Malvaceae); Hibiscus rosa-sinensis L. (Malvaceae); Hippophae salicifolia D. Don (Elaeagnaceae); Leucaena glauca (L.) Benth. (Leguminosae); Lonicera ciliosa Poir. (Caprifoliaceae); Lupinus termis Forsk. (Leguminosae); Malvaviscus conzattii Greenm. (Malvaceae); Momordica charantia L. (Curcurbitaceae); Ocimum sanctum L. (Labiatae); Prunus emarginata Walp. (Rosaceae); and Withania somnifera (L.) Dunal (Solanaceae). A large number of plants have been randomly selected and screened for spermicidal activity "in vitro" and several seem promising. Those species found to be active and the nature of the active principle(s), when known, are presented in a table as are plant-derived chemical substances of known or partially known structure reported to be spermicidal "in vitro." Plants warrant systematic study as potential sources of sperm-agglutinating compounds. Of 1600 Indian plants tested, 90 showed positive semen coagulating properties. There seems to be a lack of correlation among experimental results obtained by different groups of investigators, between data obtained "in vitro" and "in vivo," and between experimental results and information found in folklore. Factors complicating the adequate assessment of plants affecting male fertility are inadequate numbers of vehicle-treated controls, poor experimental design, problems related to insolubility of crude plant extracts, variation in routes of administration, diversity in reproductive function and control among various laboratory species, and problems in identifying plant names consistently.
( PLANTAS QUE INHIBEN LA PRODUCCION DE ESPERMA O MATAN LA MISMA UNA VEZ QUE ESTAN EN LA VAGINA)


Ned Tijdschr Geneeskd. 1977 Dec 10;121(50):1996-9.
Related Articles, Links

[Dieffenbachia: from arrow poison to house plant]

[Article in Dutch]

Van Heijst AN, Pikaaren SA, Van Kesteren RG.

En resumen, no es atoxica la planta, es de cuidado, pero no de exagerar para nada, y el paciente con alergias reiteradas debera cuidarse mas...

 

Re: OJO PLANTA MUY VENENOSA!

y yo que ya iba a coger el diccionario de inglés ... gracias Jose Andrés.

 

Re: OJO PLANTA MUY VENENOSA!

RESPONDIENDO A TODAS LAS OPINIONES VERTIDAS EN ESTE FORO, SOBRE LA TOXICIDAD DE ESTAS PLANTAS, TRANSCRIBO UN PÁRRAFO QUE EXPLICA POR QUE ES TOXICA "Las aráceas (Zamioculca, Dieffenbachia, Spathicarpa, Philodendron, Caladium, Syngonium, Aglaonema, Zantedeschia, Amorphophallus, Peltandra, Asarum, Arum, Arisaema, Alocasia, Colocasia, Pistia, etc. ). poseen cristales de rafidio ondulados de oxalato de calcio presentes en células especializadas, y con químicos asociados que causan irritación en boca y garganta si son ingeridos. Compuestos cianogénicos muchas veces presentes, y a veces con alcaloides. Muchas veces con laticíferos, canales de mucílago, o canales de resina, y látex acuoso o lechoso(Wikipedia)
Pricipio activo: OXALATO DE CALCIO
L-ASPARAGINA
Toxicidad: Mediana-Su savia resulta irritante debido a la presencia de estos elementos que aparecen en forma de cristales y que dañan los tejidos de la piel por contacto. Tiene además propiedades vomitivas e inflamatorias, su ingestión puede dañar los riñones, arterias, estómago, y en contacto con los ojos puede producir seguera temporal.
Requiere atenciòn mèdica inmediata.(información más extensa - www.botanical-on line.com)
Hojas y tallos contienen grandes cantidades de oxalato de calcio, esta planta tambien es llamada caña del mudo porque quien la muerde queda privado del habla por inflamació e irritación de la lengua, que puede llegar a provocar asfixia.(Enciclopedia Encarta)
Alguno nombres populares de araceas:
Hoja elegante - costilla de adan - potus - filodendro- taro-
Tambien se puede conocer por Internet que algunas variedades de estas plantas son comestibles, y fueron fuente de alimento para las comunidades amazonicas. Para su ingesta debe seguirse un procedimiento especifico de cocción para anular su toxina.
Del conocimiento adquirido en estos días sobre estas plantas, puedo emitir una opinion en el sentido que si bien es una planta peligrosa, puede ser manipulada con los cuidados necesarios. Pero esto también me lleva a pensar "Que necesidad tengo de poner en riesgo a mi familia por tener una planta como esta? Leí algunos cometarios que decian "pero quien se va a comer una planta, o todo es una mentira", solo puedo contestarle a estas persona que las plantas en general tienen elementos químicos cuya ingesta directa puede ser nociva para las personas y animales, aún siendo alimentos que se consumen habitualmente. Con respecto a las plantas ornamentales o de jardín, los niños tiene la tendencia a jugar con ellas, pellizcandolas, o cortandolas para hacer la comidita en el juego de la visita, si Uds. no tiene niños, recuerden su infancia.

 

Re: OJO PLANTA MUY VENENOSA!

vamos a ver, en cada pais hay un idioma comun, ¿porque insistis en el ingles si estamos en España? para que no nos enteremos todos supongo.
que cada uno haga lo que le parezca pero si no le importa lo que aqui se dice para que entra a malmeter y llamarnos mentirosos (de forma educada) pues no entreis y dejarnos a los demas compartir conocimientos. A algunos de nosotros nos interesa saber del tema ¿podriais dejar que nos esplayemos agusto? Gracias de antemano.

 

Re: OJO PLANTA MUY VENENOSA!

Mariadelosangeles, creo que tienes razon, una cosa es que en casa todos seamos mayores y otra que haya pequeñuelos.
Si sabes de mas plantas comunes me gustaria saber de ellas, tengo curiosidad por saber del tema. Si ahora no hay pequeños en casa es posible que algun dia los haya.

 

Re: OJO PLANTA MUY VENENOSA!

A la fruta tambien le das lejia, querido Pepemel? pos yo creia que era solo a la lechuga unas gotitas cuando la pones en remojo...Jo pos que confia que soy yo...que a los tomates apenas les enseño el grifo...! muy mal !...¿verdad?

Nuriya...yo esos relatos en ingles, como mi ingles es basico pues tampoco los puedo leer...

 

Re: OJO PLANTA MUY VENENOSA!

yo una vez la matiqe y me pico toda la lengua

 

Re: OJO PLANTA MUY VENENOSA!

Hola saludos a todos los foristas

Este asunto de la plantita es muy importante y en vista de la cantidad de opiniones en uno y otro sentido decidí realizar mi propia indagación y descubrí que definitivamente NO ES CONVENIENTE TENERLAS DENTRO DE CASA.
Aquí les doy 2 enlaces que mustran claramente el grado de toxicidad y por ende de peligrosidad de la dichosa planta:
http://tratado.uninet.edu/c101101.html
http://pericosaustralianos.mforos.com/1487016/6999834-informacion-util-plantas-venenosas-para-los-periquitos/

También conseguí una excelente información en un libro sobre cuidados de emergencias, parte del cual transcribo a continuación:

UNINET http://tratado.uninet.edu/c101101.html

Sociedad Andaluza de Medicina Intensiva y Unidades Coronarias (SAMIUC)

Tratado de Medicina Intensiva

Urgencias, Emergencias y Cuidados Críticos

Capítulo 10. 11. Intoxicación por plantas y setas

1. INTOXICACION POR PLANTAS


La ingesta de plantas produce entre el 1-2 % de todas las intoxicaciones; la mayoría
son accidentales y en el 85 % de los casos afectan a niños menores de 6 años.


...la habitual falta de conocimientos de botánica por parte de los médicos, suele di-
ficultar la identificación de la planta y la valoración de su potencial toxicidad.
Contribuye a ello la existencia de diversos nombres populares y locales que aportan
los pacientes y que no suelen figurar en los libros junto al nombre botánico.



1. 1. PLANTAS CON AFECTACIÓN GASTROINTESTINAL

Es sin ninguna duda el síndrome más frecuente. Su intensidad puede variar desde una
ligera irritación de la mucosa oral a un severo cuadro gastroenterítico. Las diarreas
y más especialmente los vómitos, pueden reflejar en muchas ocasiones simplemente la
intolerancia local a un producto extraño e irritante y contribuyen a la expulsión del
tóxico. Si estos no se produjeran, deben considerarse el vaciado gástrico y la administración de carbón activado.


Existe un grupo de plantas que contienen oxalatos; algunas de ellas son utilizadas como
alimentos o purgantes, como el ruibarbo o Rheum rhaponticum y las acederas, Rhumex acetosa. Esta última, muy apreciada para la elaboración de sopas, puede ser confundida con
sus congéneres Rhumex conglomeratus y Rhumex crispus, que son mucho más ricas en ácido oxálico, en especial la segunda. Precisamente por ingestión de esta falsa acedera se registró un caso mortal a finales de los ochenta en Barcelona. Otras plantas con oxalatos son decorativas, como las de la familia Araceae, Dieffenbachia, Philodendron, Caladium, Alocasia, y están presentes en numerosos hogares y lugares comunes.

La ingesta abundante de las consideradas comestibles puede provocar alteración hepática,
renal y cardíaca por depósito de cristales de oxalato cálcico insolubles, formados tras
la reacción del ácido oxálico absorbido con el calcio plasmático; los casos graves se
acompañan de hipocalcemia y acidosis metabólica. El tratamiento es sintomático y de mantenimiento. Por otro lado, el solo intento de masticación de hojas de aráceas decorativas da lugar a irritación orofaríngea, disfagia, náuseas, vómitos y, en casos extremos puede aparecer shock. Para la irritación orofaríngea típica de la Dieffenbachia (caña muda) es
útil el dar a chupar trocitos de hielo.

Tal y como se puede leer en este extracto del libro es conocida de vieja data la toxicidad de la Dieffenbachia (caña muda) por lo que en lo que a mí respecta está claro que hay que sacarla del interior de nuestras casas pues los niños y las mascotas pueden accidentalmente envenenarse con ella.

De modo que como reza el refrán: "precaución no es cobardía"...

Esta es mi opinión.

Eudimarcita desde la República Bolivariana de Venezuela

 

Re: OJO PLANTA MUY VENENOSA!

si tienes la tendencia a comerte las plantas de interior y ornamentales mejor no tengas ninguna, pues todo lo que no se come es porq no es comestible! es decir todas las plantas ornamentales pueden ser toxicas, en mayor o menor grado! en mi casa hubo una durante mi infancia y la de mi hermana, hasta que unos 20 años despues mi perro la destruyo siendo cachorro y ademas del regaño apoteosico que se gano y algo de diarrea, no le paso nada mas.

la toxicidad muchas veces dependera de las reacciones alergicas de cada quien, y es importante ecordar, las `plantas que no forman pare de nuestras ensaladas. no lo hace porque NO son comestibles!

cuantos de nosotros no crecimos con esa planta en casa???
alguien conoce de manera directa a alguien que se haya envenenado con ella???
esto es una leyenda urbana!!!!!!!!

una pobre planta no tiene porque pagar por la ociosidad de alguien que envio un correo electronico!!!!

sencillamente ninguna planta ornamental debe ser masticada!!!!!

 

Re: OJO PLANTA MUY VENENOSA!

esta planta es la que mas le gusta a mi madre y tiene un montón de plantas de diferentes variantes y especies, y no es tan mala como la gente dice, y en mi país se le dice lotería, luego les cuento como se le buscan los números de la lotería a esta mata. si quieres jugar lotería y ganar.
esta mata casi todas las casas en costa rica tienen al menos una.

 

Re: OJO PLANTA MUY VENENOSA!

Déjate de rollos, que el hilo ya está agotado.

 

Re: OJO PLANTA MUY VENENOSA!

HOLA HE INGRESADO AL FORO PUES HE SIDO VICTIMA SI SE PUEDE ASÍ DECIR DE UNA PLANTA VENENOSA.
HOY DOMINGO 11 DE SEPTIEMBRE DE 2011 A ESO DE LAS 10 Y 30 DE LA MAÑANA TUVE UN INCIDENTE CON ESTA PLANTA ORNAMENTAL VENENOSA.
NO SE POR QUÉ RAZÓN SE ME OCURRIÓ DESPUÉS DE CORTAR VARIOS TROZOS, PROBARLA Y ME LA LLEVÉ A LA BOCA PENSANDO QUE POR LO MENOS SABRÍA FEO Y LA BOTARÍA O LA ESCUPIRÍA SI FUESE MUY FUERTE EL SABOR. UNA VEZ LA MEDIO RASPÉ CON LOS DIENTES PARA SABOREARLA NO SENTÍ NINGÚN SABOR
PERO INMEDIATAMENTE SENTÍ UN ARDOR INMENSO EN TODA LA BOCA Y GARGANTA, TANTO ASÍ QUE ME FUI CORRIENDO AL LAVAMANOS PARA JUAGARME LA BOCA CON ABUNDANTE AGUA, PERO EL ARDOR NO SE QUITABA, CASI NO PODÍA TRAGAR EL AGUA Y COMENZÓ EL ORGANISMO COMO A DEFENDERSE DEL VENENO CON ABUNDANTE SALIVA ESPESA. MI ESPOSA ESTABA CONMIGO Y RECORDÓ QUE HABÍA VISTO UN ARTICULO EN INTERNET DONDE HABLABAN ACERCA DE ESTA PLANTA VENENOSA. GRACIAS A ESTE ARTICULO SUPIMOS DE QUE SE TRATABA HE INMEDIATAMENTE FUIMOS A URGENCIAS DONDE ME APLICARON ANTIINFLAMATORIOS Y ANTIALÉRGICOS.
HA SIDO UN DÍA MUY DURO QUE NO LO DESEO PARA NADIE.
TODO ESTO POR IGNORAR QUE EXISTEN PLANTAS CON TANTO PODER TOXICO.
RECOMIENDO NO TENERLAS CERCA DE LOS NIÑOS.
AL PARECER TODAS LAS PLANTAS DE ESTA ESPECIE SON VENENOSAS.
SON LAS 20 Y 38 Y AÚN ME SIENTO CON LA LENGUA PESADA, ME ARDEN LOS LABIOS Y TENGO UN DOLOR EN EL PECHO. PERO AFORTUNADAMENTE NO FUE MUCHO LO QUE ALCANCÉ A PROBAR Y YA ME SIENTO MEJOR.